2014年1月27日星期一

Why IgA Nephropathy Patients Get Hypertention Nephropathy

IgA Nephropathy is the most common of glomerulonephritis in the world, which is characterized by predominant IgA deposition in the glomerular messangium. With the development of illness condition, some IgA Nephropathy patients get Nephrotic Hypertention.

With the progression of IgA Nephropathy, the renal intrinsic cells can be damaged and kidney dysfunction occurs. Water and sodium retention appears, so the kidney blood vessels are compressed, and blood flow decreases. Then renal receptor will secrete more renin, and then angiotensin concentration increases, which make blood vessel shrink, thus increasing the blood pressure. High blood pressure and renal injury may aggravate each other. In general, if IgA Nephropathy patients get hypertension, their renal function is about to be injured has already been injured.

Once patients of IgA Nephropathy have symptoms of Nephrotic Hypertension, they should receive effective treatment instead of self-medication. IgA Nephropathy led hypertension is different from primary hypertension. The key of treating hypertension is to treat IgA Nephropathy through methods of delaying or blocking renal fibrosis. Only in this way, can we treat hypertension radically.

The Hot Compress Therapy of Huaxia Kidney Disease Hospital can not only treat IgA Nephropathy from the root by degrading the IgA deposits but also restore the renal function. Once injured kidney is recovered, IgA Nephropathy can be relieved and hypertension caused by IgA Nephropathy can also be relieved.

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